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2020/8/27 10:31:22 | Browse: 464 | 下载: 631
| Publication Name |
世界华人消化杂志 |
| 手稿编号 |
37779 |
| 手稿来源省、自治区或特别行政区 |
湖北省 |
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Received |
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2020-05-25 00:32 |
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Peer-Review Started |
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2020-05-27 00:43 |
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To Make the First Decision |
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Return for Revision |
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2020-05-29 18:33 |
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修回稿 |
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2020-06-05 14:41 |
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Second Decision |
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2020-07-03 11:07 |
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期刊总编辑接受 |
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公司总编辑接受 |
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2020-07-04 23:04 |
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预出版 |
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2020-07-04 23:04 |
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缴纳版面费 |
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Edit the Manuscript by Language Editor |
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排版 |
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2020-08-27 08:40 |
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在线出版 |
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2020-08-27 10:31 |
| ISSN |
1009-3079 (print) and 2219-2859 (online) |
| 开放获取 |
This article is an open-access article that was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution NonCommercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/ |
| 版权 |
© The Author(s) 2020. Published by Baishideng Publishing Group Inc. All rights reserved. |
| Article Reprints |
For details, please visit: http://www.wjgnet.com/bpg/gerinfo/247
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| Permissions |
For details, please visit: http://www.wjgnet.com/bpg/gerinfo/207
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| Publisher |
Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA |
| 网址 |
http://www.wjgnet.com |
| 专业类型 |
消化内科学 |
| 手稿栏目类型 |
文献综述 |
| 文章标题 |
热休克蛋白5与炎症性肠病研究进展
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| 手稿来源 |
自由来稿 |
| 所有作者列表 |
高菲, 范恒 |
| ORCID |
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| 基金项目及其编号 |
| 基金项目名称 |
基金编号 |
| 国家自然科学基金资助项目 |
8190141912 |
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| 通讯作者 |
范恒, 教授, 主任医师, 430022, 湖北省武汉市解放大道1277号, 华中科技大学同济医学院附属协和医院中西医结合科. fanheng009@aliyun.com |
| 关键词 |
热休克蛋白5; 炎症性肠病; 内质网稳态; 细胞凋亡; 炎症反应 |
| 核心内容提要 |
炎症性肠病(inflammatory bowel disease, IBD)发生时, 肠上皮细胞内质网过度应激, 热休克蛋白5(heat shock protein 5, HSPA5)的致细胞凋亡作用远大于其保护细胞的作用, 因此, 可以将过表达的HSPA5看作是IBD肠上皮细胞存活的危险因素. |
| 出版日期 |
2020-08-27 10:31 |
| 引文著录 |
高菲, 范恒. 热休克蛋白5与炎症性肠病研究进展. 世界华人消化杂志 2020 |
| URL |
https://www.wjgnet.com/1009-3079/full/v28/i16/802.htm |
| DOI |
https://dx.doi.org/10.11569/wcjd.v28.i16.802 |
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